IJCCR 2013, Vol.3, No.2, 7
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TSCS also do not have any alveoli performance
enhancing role {which in asthma are intact}. Long
period field experience of juxtaposing severe weather
events with problems of the patients having chest
diseases, indicates, that, the otherwise cheerful patient
is invariably lead into crisis, post pass of the cyclone.
SOS is thence the only relief out. And, SOS is
unavailable in the rural of the remote. Long period
observation had to be adhered to.
We noted, that in the wards of all the hospitals in the
severe cyclone effected regions, post pass of a weather
system, all cardio-thoracic in-patients experience
heightened bouts of cough and seizure {idiopathic} as
the atmospheric pressure returns to 1 000 hPa, with
preponderance around mid-night of the local winter
period {numerous pass away}, and particularly post
the zenithal pass of the sun/moon during new/full
moon period. Even asthmatic clinicians and the
meteorologists get afflicted as much seriously and
know not why and how? Often it is misunderstood as
mass affliction, community problem, radical steps are
called for. Our study indicates that, at zenithal location
(
new/full moon), the astrals impart maximum
buoyancy to the atmospheric fluid column (greater
height dimension), which imparts boundary\resistance
effect. Post zenithal pass the buoyant fluid is
unleashed as ‘gravity waves’ (Niranjan Kumar and
Ramkumar, 2008). Gravity waves of greater
magnitude are more associated with depression/
cyclone pass. They are intense and have grave
prognosis. Because maximum number of casualties.
Over two decades we followed such nature based
inspiration.
Moreover, when the lower lungs of a chronic
asthma/COPD patient inflates beyond the daily
average {restoration of normal atmospheric pressure},
it also means higher diastole (higher back pressure on
the heart), which we know is fraught with danger.
There is also a concurrent higher systole. As the
atmospheric pressure switches back to normalcy, the
patient starts indicating persistent cough, wheeze,
right ventricle stress, perspiration, and complications.
SOS becomes necessary. Therefore, it may be
inadvisable to introduce an alveoli performance
enhancer. Bronchodilators are here to stay. Atmospheric
low systems ingresses deep inland, hence SOS may
become necessary in greater regions of India. If a
COPD patient is quarantined in a room having a
barometric pressure ranging between 960~980 with
moist oxygen, then she/he will revive early. The long
term panacea is breathing exercises, on and often
through the day, on sustained basis {Gold standard}.
Hindu apex lexicon the Sabdakalpadrum cites Hema-
chandra the grammarian of yore to indicate that
SWASA (popular call name of asthma) means
air-flow and again cites Rajnirghanta another linguist
of yore, who qualifies it with the term ‘life’, so it
amounts to SWASA=‘life air flow’ (Sabdakalpadrum,
1886).
In 1892, William Osler
(www.collectionscana-
da.gc.ca) suggested that inflammation played an
important role in asthma. Histamins are part of
physiological defense mechanism primarily associated
with (contraction) the smooth muscles of the
respiratory systems. Allergens cause heightened
release of bronchial histamine receptors. Results in
efficient and intensified uptake of Histamins (1 & 2)
causes inflammation and also in vessel tonus
(
constriction in general), It results in influx of Ca
+
which increases force of contraction of arterial and
ventricular muscles. Histamins also trigger Mast cells
from the lungs sub-mucosa (Arrang et al., 1983).
Histamin trigger is also via a loop. Thence a
deep-seated inflammatory response is initiated. As the
cyclone builds up surface air flows towards the core of
the cyclonic system. We noted allergens flow away.
There is ‘fresh’ feel in the air, even by the
non-asthmatic. This is one more underlying cause of
‘
feel good’.
Over the period we also noted anoxia was\is not being
efficiently addressed by the breathing apparatus with
any amount of tweaking the O
2
inflow; or altering the
patient’s posture. There is no anisocytosis, yet there is
pulmonary distress with mental and cerebral
disorientation and disregulation; with slack\slumped
neck and warm- non perspiring nape. We gravitated to
the point that anoxia was response pathology
{
bio-chemical} triggered by the used gas over-loaded
alveoli (un-expirated gas). Among the deficiency was
K
+
{
brain has a very narrow range for it}. In
continuum of such status of K
+
deficiency, rapid onset
of the mechanical component of the malady manifests,
with a pronounced steep sinking, thereafter. Cortico-
steroids, we theorised were either {i} up-regulating K
+
availability and\or {ii} becausing a heightened